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It is a dual glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) receptor agonist, which means it stimulates insulin secretion and inhibits glucagon release in a glucose-dependent manner
The two medications have distinct mechanisms tolerance of one does not predict tolerance of the other
GIP receptor agonism adds incremental insulin sensitization in adipose tissue and may improve GI tolerability relative to pure GLP-1 agonism at equivalent doses, though the clinical net effect on tolerability varies by individual [6]
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